APICALL:
https://apimlqv2.tenwiseservice.nl/api/mlquery/refset/citations/?pmids=32324828,32324857&apikey=YOUR_APIKEY
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RESULT: 
{
    "result": {
        "citations": [
            {
                "abstract": "Vascular deficits are a fundamental contributing factor of diabetes-associated diseases. Although previous studies have demonstrated that the pro-angiogenic phase of wound healing is blunted in diabetes, a comprehensive understanding of the mechanisms that regulate skin revascularization and capillary stabilization in diabetic wounds is lacking. Using a mouse model of diabetic wound healing, we performed microCT analysis of the 3-dimensional architecture of the capillary bed. As compared to wild type, vessel surface area, branch junction number, total vessel length, and total branch number were significantly decreased in wounds of diabetic mice as compared to WT mice. Diabetic mouse wounds also had significantly increased capillary permeability and decreased pericyte coverage of capillaries. Diabetic wounds exhibited significant perturbations in the expression of factors that affect vascular regrowth, maturation and stability. Specifically, the expression of VEGF-A, Sprouty2, PEDF, LRP6, Thrombospondin 1, CXCL10, CXCR3, PDGFR-\u03b2, HB-EGF, EGFR, TGF-\u03b21, Semaphorin3a, Neuropilin 1, angiopoietin 2, NG2, and RGS5 were down-regulated in diabetic wounds. Together, these studies provide novel information about the complexity of the perturbation of angiogenesis in diabetic wounds. Targeting factors responsible for wound resolution and vascular pruning, as well those that affect pericyte recruitment, maturation, and stability may have the potential to improve diabetic skin wound healing.",
                "authors": "Uzoagu A Okonkwo;Lin Chen;Da Ma;Veronica A Haywood;May Barakat;Norifumi Urao;Luisa A DiPietro",
                "chemicalterms": "",
                "issue": "4",
                "journal_title": "PLoS One",
                "meshterms": "Animals;Blood Vessels;Capillaries;Diabetes Mellitus, Experimental;Female;Mice;Mice, Inbred C57BL;Neovascularization, Pathologic;Pericytes;Permeability;Wound Healing;X-Ray Microtomography",
                "page": "e0231962",
                "pmid": "32324828",
                "pubmed_date": "2020-04-24",
                "title": "Compromised angiogenesis and vascular Integrity in impaired diabetic wound healing.",
                "volume": "15"
            },
            {
                "abstract": "Purpose: Geographic atrophy (GA) secondary to age-related macular degeneration (AMD) is a progressive disease with no treatment option. Previous studies show chemokine-mediated recruitment of immune cells in the retina, and therefore we investigated systemic levels of chemokines and chemokine receptors in patients with GA. Methods: This observational prospective study was conducted at a single center. We included 122 participants with no immune disease: 41 participants with GA and no choroidal neovascularization, 51 patients with neovascular AMD, and 30 healthy control individuals. Flow cytometric analysis was used to detect expression level of C-C chemokine receptor (CCR)1, CCR2, CCR3, CCR5, and C-X-C motif chemokine receptor (CXCR)3 on peripheral blood mononuclear cells (CD14+ monocytes, CD4+ T cells, CD8+ T cells). Plasma levels of C-C motif ligand (CCL)11, C-X-C motif chemokine (CXCL)10, and CCL5 were measured by specific immunoassays. Enlargement rate of GA lesion was measured from autofluorescence images. Results: Participants with GA have a specific chemokine profile with a higher expression of CCR5 than healthy controls in peripheral blood mononuclear cells, and a higher plasma levels of CCL-5. Further, GA was associated with higher monocytic expression of CCR2 than in neovascular AMD. We found that a high expression level of CCR5 on CD8+ T cells was associated with slower enlargement rate of atrophic lesion. Conclusions: The study showed an association between systemic chemokine profile and GA formation. Further studies are needed to fully elucidate the possible role of systemic chemokine regulation in mediating pathogenesis of GA.",
                "authors": "Marie Krogh Nielsen;Yousif Subhi;Christopher Rue Molbech;Mads Kr\u00fcger Falk;Mogens Holst Nissen;Torben Lykke S\u00f8rensen",
                "chemicalterms": "CCR5 protein, human;Chemokine CCL5;Receptors, CCR5;Vascular Endothelial Growth Factor A",
                "issue": "4",
                "journal_title": "Invest Ophthalmol Vis Sci",
                "meshterms": "Aged;Case-Control Studies;Chemokine CCL5;Choroidal Neovascularization;Female;Gene Expression Regulation;Geographic Atrophy;Humans;Leukocytes, Mononuclear;Male;Prospective Studies;Receptors, CCR5;Reference Values;Sensitivity and Specificity;Severity of Illness Index;Vascular Endothelial Growth Factor A;Wet Macular Degeneration",
                "page": "28",
                "pmid": "32324857",
                "pubmed_date": "2020-04-24",
                "title": "Chemokine Profile and the Alterations in CCR5-CCL5 Axis in Geographic Atrophy Secondary to Age-Related Macular Degeneration.",
                "volume": "61"
            }
        ]
    },
    "status": "Success"
}